contractile protein
- 收缩蛋白;收缩蛋白质
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The presence of a contractile protein in higher plants
高等植物中的收缩蛋白
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Contractile protein expression , phenotypic modulation and proliferation of vascular smooth muscle cells in primary culture
培养血管平滑肌细胞收缩蛋白表达与表现型转变及细胞增殖的关系
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Changes in Contractile Protein , Sarcoplasmic Reticulum ATPase in Mice Cardiac Muscle with Aging and Effects of Swimming
小鼠心肌收缩蛋白、肌浆网ATP酶的增龄性变化及游泳训练对其的影响
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This increased smooth muscle mass is related either to the abnormal bronchial smooth muscle cell proliferation or the accumulation of contractile protein .
增多的气管平滑肌物质或与支气管平滑肌细胞的异常增殖有关,或与收缩蛋白聚积有关。
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A large portion of the cytoskeleton consists of threadlike microfilaments composed mainly of the contractile protein actin .
细胞骨架大部分由微丝组成,微丝主要由可收缩的肌动蛋白组成。
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CONCLUSION : MCI-154 directly enhances the Ca2 + sensitivity of contractile protein but has little effect on Ca2 + release from SR in rat skinned cardiac fibers .
结论:MCI-154直接增强心肌收缩蛋白的Ca~(2+)敏感性,但对SR的Ca~(2+)释放无明显作用。
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In reference to mechanisms of peripheral muscle fatigue , the possible parts in which fatigue may happen were introduced respectively from the neuromuscular junction to the contractile protein of skeletal muscle .
有关外周疲劳机制,分别介绍神经肌肉接头至骨骼肌收缩蛋白等发生疲劳的部位。
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Sports cardiac hypertrophy is a response of cardiac myocytes to various exercise induced stimuli such as mechanical overload , humoral factors which lead to genetic expression changes in cardiac contractile protein genes .
运动性心肌肥大是心肌细胞对运动诱发的各种刺激因素如机械负荷、体液因子以及由这些因素导致的心肌收缩蛋白基因表达改变的功能反应。
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Conclusion The sensitivity o myocardial contractile protein to calcium is decreased in the rats with endotoxic rats . MCI-154 , a calcium sensitizer , is able to reverse the decreased sensitivity significantly and increase the Tmax of the myocardium .
结论大鼠内毒素休克后,心肌收缩蛋白对钙的敏感性降低,MCI154可明显增加内毒素休克大鼠心肌收缩蛋白对钙的敏感性,增加心肌钙最大激活张力。
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Conclusion 17 β - estradiol inhibits hypertrophy of cardiomyocytes and its mechanisms might be related with the suppression of c-fos protein expression , the reversion of the embryonic switching of contractile protein genes (β MHC and α - skA ), the increase of ANP mRNA expression .
结论17β-雌二醇可抑制心肌细胞肥大,其作用机制可能与抑制原癌基因c-fos的蛋白表达,逆转收缩蛋白基因(β-MHC和α-skA)向胚胎型转化及促进ANPmRNA表达有关。